The Roberson Lab
The Roberson Lab

Publications

 

Dr. Roberson’s Online Bibliographies

 

Citations per year

Source: Google Scholar
 

Featured Papers

(click on journal name for link to paper)

Publications Gallery
Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability
Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability

Voskobiynyk, Y., J.R. Roth, J.N. Cochran, T. Rush, N.V.N. Carullo, J.S. Mesina, M. Waqas, R.M. Vollmer, J.J. Day, L.L. McMahon, and E.D. Roberson. (2020). Alzheimer’s disease risk gene BIN1 induces Tau-dependent network hyperexcitability. eLife 10.7554/eLife.57354.

Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutations
Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutations

Arrant, A.E., J.R. Roth, N.R. Boyle, S.N. Kashyap, M.Q. Hoffmann, C.F. Murchison, E.M. Ramos, A.L. Nana, S. Spina, L.T. Grinberg, B.L. Miller, W.W. Seeley, and E.D. Roberson. (2019). Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutations. Acta Neuropathol. Commun. 7:218

Neurodegenerative Disease–Associated Variants in TREM2 Destabilize the Apical Ligand-Binding Region of the Immunoglobulin Domain
Neurodegenerative Disease–Associated Variants in TREM2 Destabilize the Apical Ligand-Binding Region of the Immunoglobulin Domain

Dean, H.B., E.D. Roberson, and Y. Song. (2019). Neurodegenerative Disease–Associated Variants in TREM2 Destabilize the Apical Ligand-Binding Region of the Immunoglobulin Domain. Front. Neurol. DOI: 10.3389/fneur.2019.01252.

Progranulin gene therapy improves lysosomal function and microglial pathology associated with frontotemporal dementia and neuronal ceroid lipofuscinosis
Progranulin gene therapy improves lysosomal function and microglial pathology associated with frontotemporal dementia and neuronal ceroid lipofuscinosis

A.E. Arrant, V.C. Onyilo, D.E. Unger, and E.D. Roberson. (2018). J. Neurosci. 38:2341–2358.

Commentary in Nat. Rev. Neurology

Restoring neuronal progranulin reverses deficits in a frontotemporal dementia model
Restoring neuronal progranulin reverses deficits in a frontotemporal dementia model

A.E. Arrant, A.J. Filiano, D.E. Unger, A.H. Young, and E.D. Roberson. (2017). Brain 140:1447–1465.

Tau-dependent Kv4.2 depletion and dendritic hyperexcitability in a mouse model of Alzheimer’s disease
Tau-dependent Kv4.2 depletion and dendritic hyperexcitability in a mouse model of Alzheimer’s disease

Hall, A.M., B.T. Throesch, S.C. Buckingham, S.J. Markwardt, Y. Peng, Q. Wang, D.A. Hoffman, and E.D. Roberson. (2015). Tau-dependent Kv4.2 depletion and dendritic hyperexcitability in a mouse model of Alzheimer’s disease. J. Neurosci. 35:6221–6230

Tau-mediated NMDA receptor impairment underlies dysfunction of a selectively vulnerable network in a mouse model of frontotemporal dementia
Tau-mediated NMDA receptor impairment underlies dysfunction of a selectively vulnerable network in a mouse model of frontotemporal dementia

Warmus, B.A., D.R. Sekar, E. McCutchen, G.D. Schellenberg, R.C. Roberts, L.L. McMahon, and E.D. Roberson. (2014). J. Neurosci. 34:16482–16495.

Reducing endogenous tau ameliorates Aβ-induced deficits in an Alzheimer’s disease mouse model
Reducing endogenous tau ameliorates Aβ-induced deficits in an Alzheimer’s disease mouse model

Roberson, E.D., K. Scearce-Levie, J.J. Palop, F. Yan, I. Cheng, T. Wu, H. Gerstein, G.-Q. Yu, and L. Mucke. (2007). Science 316:750–754.

Commentary in N. Engl. J. Med.

Commentary in Nat. Rev. Neurosci.

Commentary on AlzForum